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Mutagenesis Advance Access originally published online on August 8, 2006
Mutagenesis 2006 21(5):305-311; doi:10.1093/mutage/gel036
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© The Author 2006. Published by Oxford University Press on behalf of the UK Environmental Mutagen Society. All rights reserved. For permissions, please email: journals.permissions@oxfordjournals.org

Organ-targeted mutagenicity of nitrofurantoin in Big Blue transgenic mice

Philippe Quillardet1, Xavier Arrault3, Valérie Michel2 and Eliette Touati2,*

1 Unité des Cyanobactéries, Institut Pasteur 28, Rue du Dr Roux, 75724 Paris Cedex15, France 2 Unité de Pathogénie Bactérienne des Muqueuses, Institut Pasteur 28, Rue du Dr Roux, 75724 Paris Cedex15, France 3 Service de Pharmacie Clinique et des Biomatériaux, Groupe Hospitalier Bichat-Claude Bernard AP-HP, 46, Rue Henri Huchard, 75877 PARIS Cedex 18, France

Nitrofurans are widely used in human medicine, as nitrofurantoin and nifuroxazide, still prescribed for long-term antimicrobial prophylaxis of urinary tract and gastrointestinal infection in humans respectively. Recent experiments in mammals, as well as reports mentioning toxic effects in humans associated with a long-term use, specially in the case of nitrofurantoin, raised the need for reevaluating their genotoxicity. The objective of this study was to determine whether these two compounds induce a mutagenic effect in the Big Blue transgenic mouse mutation assay. Mice were orally treated either with nitrofurantoin or nifuroxazide for five consecutive days and sacrificed 3 weeks later. In order to optimize the genotoxic response, the doses used for each compound were 25-fold higher as the posology in humans. They corresponded to 50% of the highest doses tolerated by mice. The mutant frequency was determined from kidney, lung, bladder, caecum, colon, small intestine, spleen and stomach. A weak mutagenic response of nitrofurantoin-treated mice specifically in the kidney was observed. As in the case of other nitrofuran compounds, the mutation spectra determined from treated samples exhibited slightly more GC->TA transversions as compared with untreated conditions. These data are relevant to the targeted action of nitrofurantoin as a urinary antimicrobial agent. No significant increase of mutants was detected in the case of nifuroxazide-treated mice whatever the organs analysed.

*To whom correspondence should be addressed. Tel: +33 1 40 61 37 85; Fax: +33 1 40 61 36 40; Email: etouati{at}pasteur.fr


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